Перегляд за автором "Stepanenko, A.A."

Сортувати за: Порядок: Результатів:

  • Cisplatin treatment of C6 rat glioma in vivo did not influence copy number alterations and growth pattern of tumor-derived resistant cells 
    Stepanenko, A.A.; Baklaushev, V.P.; Vassetzky, Y.S.; Dmitrenko, V.V. (Вiopolymers and Cell, 2015)
    Aim. To investigate whether the cisplatin treatment of C6 rat glioma in vivo impacts the copy number alterations (CNAs), proliferation and colony formation efficiency (CFE) of tumor-derived cisplatin-resistant cells. ...
  • Evolutionary karyotypic theory of cancer versus conventional cancer gene mutation theory 
    Stepanenko, A.A.; Kavsan, V.M. (Вiopolymers and Cell, 2012)
    Согласно общепризнанной генной теории рака, в течение последних десятилетий рак считали генным заболеванием, возникающим в результате детерминистического последовательного накопления мутаций в небольшой группе раковых ...
  • Expression of genes belonging to the IGF-system in glial tumors 
    Dmitrenko, V.V.; Kavsan, V.M.; Boyko, O.I.; Rymar, V.I.; Stepanenko, A.A.; Balynska, O.V.; Malisheva, T.A.; Rozumenko, V.D.; Zozulya, Y.P. (Цитология и генетика, 2011)
    The discrepancies arising from conflicting evidence on the results obtained by different laboratories in human gliomas are discussed. Our data highlight the importance of viewing the IGF-related proteins as a complex ...
  • From reverse transcription to human brain tumors 
    Dmitrenko, V.V.; Avdieiev, S.S.; Areshkov, P.O.; Balynska, O.V.; Bukreieva, T.V.; Stepanenko, A.A.; Chausovskii, T.I.; Kavsan, V.M. (Вiopolymers and Cell, 2013)
    Reverse transcriptase from avian myeloblastosis virus (AMV) was the subject of the study, from which the investigations of the Department of biosynthesis of nucleic acids were started. Production of AMV in grams quantities ...
  • Structure and function of oncogene-transfected immortal cells 
    Kavsan, V.M.; Kulagova, T.A.; Kuznetsova, T.A.; Semenkova, G.N.; Stepanenko, A.A.; Vassetzky, Y.S. (Вiopolymers and Cell, 2014)
    Previously we have characterized a new oncogene CHI3L1, overexpressed in the glioblastoma and the malignant 293_CHI3L1 cells, stably producing the CHI3L1 angiogenic oncoprotein. The 293_CHI3L1 cells proliferated faster and ...